project in biology

I; INTRODUCTION:
                                                           ''MUTATION"

In molecular biology and genetics, mutations are changes in a genomic sequence: the DNA sequence of a cell's genome or the DNA or RNA sequence of a virus. They can be defined as sudden and spontaneous changes in the cell. Mutations are caused by radiation, viruses, transposons and mutagenic chemicals, as well as errors that occur during meiosis or DNA replication. They can also be induced by the organism itself, by cellular processes such as hypermutation. Mutation can result in several different types of change in sequences; these can either have no effect, alter the product of a gene, or prevent the gene from functioning properly or completely. Studies in the fly Drosophila melanogaster suggest that if a mutation changes a protein produced by a gene, this will probably be harmful, with about 70 percent of these mutations having damaging effects, and the remainder being either neutral or weakly beneficial. Due to the damaging effects that mutations can have on genes, organisms have mechanisms such as DNA repair to prevent mutations. " The causes of mutations" Mutations happen for several reasons. DNA fails to copy accurately Most of the mutations that we think matter to evolution are "naturally-occurring." For example, when a cell divides, it makes a copy of its DNA — and sometimes the copy is not quite perfect. That small difference from the original DNA sequence is a mutation.

External influences can create mutations Mutations can also be caused by exposure to specific chemicals or radiation. These agents cause the DNA to break down. This is not necessarily unnatural — even in the most isolated and pristine environments, DNA breaks down. Nevertheless, when the cell repairs the DNA, it might not do a perfect job of the repair. So the cell would end up with DNA slightly different than the original DNA and hence, a mutation.

              there are 8 different kinds of syndrome            






                              progeria syndrome 

Progeria (also known as "Hutchinson–Gilford Progeria Syndrome", "Hutchinson–Gilford syndrome" and "Progeria syndrome" is an extremely rare genetic condition wherein symptoms resembling aspects of aging are manifested at an early age. The word progeria comes from the Greek words "pro" (πρό), meaning "before", and "géras" (γῆρας), meaning "old age". The disorder has very low incidences and occurs in an estimated 1 per 8 million live births. Those born with progeria typically live to their mid teens and early twenties.It is a genetic condition that occurs as a new mutation (de novo), and is rarely inherited. Although the term progeria applies strictly speaking to all diseases characterized by premature aging symptoms, and is often used as such, it is often applied specifically in reference to Hutchinson-Gilford Progeria Syndrome (HGPS). Scientists are particularly interested in progeria because it might reveal clues about the normal process of aging. Progeria was first described in 1886 by Jonathan Hutchinson.It was also described independently in 1897 by Hastings Gilford. The condition was later named Hutchinson-Gilford Progeria Syndrome (HGPS). cri-do-chat syndrome:

Cri du chat syndrome, also known as chromosome 5p deletion syndrome, 5p minus syndrome or Lejeune’s syndrome, is a rare genetic disorder due to a missing part of chromosome 5. Its name is a French term (cat-cry or call of the cat) referring to the characteristic cat-like cry of affected children. It was first described by Jérôme Lejeune in 1963.[1] The condition affects an estimated 1 in 50,000 live births, strikes all ethnicities, and is more common in females by a 4:3 ratio turner syndrome:

cTurner syndrome or Ullrih-Turner syndrome (also known as "Gonadal dysgenesis":550), 45 XO, encompasses several conditions in human females, of which monosomy X (absence of an entire sex chromosome, the Barr body) is most common. It is a chromosomal abnormality in which all or part of one of the sex chromosomes is absent (unaffected humans have 46 chromosomes, of which two are sex chromosomes). Normal females have two X chromosomes, but in Turner syndrome, one of those sex chromosomes is missing or has other abnormalities. In some cases, the chromosome is missing in some cells but not others, a condition referred to as mosaicism or 'Turner mosaicism'. Occurring in 1 in 2000 – 1 in 5000 phenotypic females, the syndrome manifests itself in a number of ways. There are characteristic physical abnormalities, such as short stature, swelling, broad chest, low hairline, low-set ears, and webbed necks. Girls with Turner syndrome typically experience gonadal dysfunction (non-working ovaries), which results in amenorrhea (absence of menstrual cycle) and sterility. Concurrent health concerns are also frequently present, including congenital heart disease, hypothyroidism (reduced hormone secretion by the thyroid), diabetes, vision problems, hearing concerns, and many autoimmune diseases. Finally, a specific pattern of cognitive deficits is often observed, with particular difficulties in visuospatial, mathematical, and memory areas. Turner's syndrome is named after Henry H. Turner. Turner syndrome or Ullrich-Turner syndrome (also known as "Gonadal dysgenesis":550), 45 XO, encompasses several conditions in human females, of which monosomy X (absence of an entire sex chromosome, the Barr body) is most common. It is a chromosomal abnormality in which all or part of one of the sex chromosomes is absent (unaffected humans have 46 chromosomes, of which two are sex chromosomes). Normal females have two X chromosomes, but in Turner syndrome, one of those sex chromosomes is missing or has other abnormalities. In some cases, the chromosome is missing in some cells but not others, a condition referred to as mosaicismor 'Turner mosaicism'. Occurring in 1 in 2000 – 1 in 5000 phenotypic females, the syndrome manifests itself in a number of ways. There are characteristic physical abnormalities, such as short stature, swelling, broad chest, low hairline, low-set ears, and webbed necks. Girls with Turner syndrome typically experience gonadal dysfunction (non-working ovaries), which results in amenorrhea (absence of menstrual cycle) and sterility. Concurrent health concerns are also frequently present, including congenital heart disease, hypothyroidism (reduced hormone secretion by the thyroid), diabetes, vision problems, hearing concerns, and many autoimmune diseases. Finally, a specific pattern of cognitive deficits is often observed, with particular difficulties in visuospatial, mathematical, and memory areas. Turner's syndrome is named after Henry H. Turner. klinefelter syndrome:

Klinefelter syndrome, 47, XXY, or XXY syndrome is a condition in which human males have an extra X chromosome. While females have an XX chromosomal makeup, and males an XY, affected individuals have at least two X chromosomes and at least one Y chromosome. Because of the extra chromosome, individuals with the condition are usually referred to as "XXY Males", or "47, XXY Males".Klinefelter's syndrome is the symptoms of the disease Seminiferous Tubule Dysgenesis As stated below this form of hypogonadism was first described by Klinefelter et al in 1942. The account given by Klinefelter came to be known as Klinefelter's syndrome as his name appeared first on the published paper, and Seminiferous Tubule Dysgenesis was no longer used. Babies born with XXY or any other karyotype including at least 1 additional X do not have Klinefelter's syndrome, as the syndrome itself only manifests after the onset of puberty, if it manifests at all. Williams Textbook of Endocrinology 8th Edition page 879 "Seminiferous tubule dysgenesis is a common cause of primary hypogonadism and male infertility. This syndrome was first defined as a clinical entity by Klinefelter and associates. The characteristic features, which first become manifest during adolescence, are gynaecomastia, a variable degree of eunuchoidism, [atrophic testes] with hyalinization of the seminiferous tubules, aggregation of the [cells], aspermatogenesis, and increased excretion of urinary gonadotropin....." In humans, 47XXY is the most common sex chromosome aneuploidy in malesand the second most common condition caused by the presence of extra chromosomes. The chromosome constitution (karyotype) exists in roughly between 1:500 to 1:1000 live male births but many of these people may not show symptoms. Other mammals also have the XXY syndrome, including mice. Principal effects include hypogonadism and reduced fertility. A variety of other physical and behavioural differences and problems are common, though severity varies and many XXY boys have few detectable symptoms. Not all XXY men develop Klinefelter's syndrome. The syndrome was named after Dr. Harry Klinefelter, who in 1942 worked with Fuller Albright at Massachusetts General Hospital in Boston, Massachusetts and first described it in the same year. Klinefelter syndrome, 47, XXY, or XXY syndrome is a condition in which human males have an extra X chromosome. While females have an XX chromosomal makeup, and males an XY, affected individuals have at least two X chromosomes and at least one Y chromosome. Because of the extra chromosome, individuals with the condition are usually referred to as "XXY Males", or "47, XXY Males". Klinefelter's syndrome is the symptoms of the disease Seminiferous Tubule Dysgenesis As stated below this form of hypogonadism was first described by Klinefelter et al in 1942. The account given by Klinefelter came to be known as Klinefelter's syndrome as his name appeared first on the published paper, and Seminiferous Tubule Dysgenesis was no longer used. Babies born with XXY or any other karyotype including at least 1 additional X do not have Klinefelter's syndrome, as the syndrome itself only manifests after the onset of puberty, if it manifests at all. Williams Textbook of Endocrinology 8th Edition page 879 "Seminiferous tubule dysgenesis is a common cause of primary hypogonadism and male infertility. This syndrome was first defined as a clinical entity by Klinefelter and associates. The characteristic features, which first become manifest during adolescence, are gynaecomastia, a variable degree of eunuchoidism, [atrophic testes] with hyalinization of the seminiferous tubules, aggregation of the [cells], aspermatogenesis, and increased excretion of urinary gonadotropin....." In humans, 47XXY is the most common sex chromosome aneuploidy in males and the second most common condition caused by the presence of extra chromosomes. The chromosome constitution (karyotype) exists in roughly between 1:500 to 1:1000 live male births but many of these people may not show symptoms. Other mammals also have the XXY syndrome, including mice. Principal effects include hypogonadism and reduced fertility. A variety of other physical and behavioural differences and problems are common, though severity varies and many XXY boys have few detectable symptoms. Not all XXY men develop Klinefelter's syndrome. The syndrome was named after Dr. Harry Klinefelter, who in 1942 worked with Fuller Albright at Massachusetts General Hospital in Boston, Massachusetts and first described it in the same year. down syndrome:

Down syndrome or Down's syndrome, (also known as trisomy 21), is a chromosomal condition caused by the presence of all or part of an extra 21st chromosome. It is named after John Langdon Down, the British physician who described the syndrome in 1866. The condition was clinically described earlier in the 19th century by Jean Etienne Dominique Esquirol in 1838 and Edouard Seguin in 1844. Down syndrome was identified as a chromosome 21 trisomy by Dr. Jérôme Lejeune in 1959. Down syndrome in a fetus can be identified through chorionic villus sampling or amniocentesis during pregnancy, or in a baby at birth. Down syndrome is a chromosomal condition characterized by the presence of an extra copy of genetic material on the 21st chromosome, either in whole (trisomy 21) or part (such as due to translocations). The effects and extent of the extra copy vary greatly among people, depending on genetic history, and pure chance. The incidence of Down syndrome is estimated at 1 per 733 births, although it is statistically more common with older parents due to increased mutagenic exposures upon some older parents' reproductive cells. Other factors may also play a role. Down syndrome occurs in all human populations, and analogous conditions have been found in other species such as chimpanzees and mice. Often Down syndrome is associated with some impairment of cognitive ability and physical growth, and a particular set of facial characteristics. Individuals with Down syndrome usually have low intelligence, such as to constitute mild to moderate intellectual disability. Many children with Down syndrome who have received family support, enrichment therapies and tutoring manage to graduate from high school and college, and are able to do paid work. The average IQ of children with Down syndrome is around 50, compared to normal children with an IQ of 100. A small number have a severe to high degree of intellectual disability. Individuals with Down syndrome may have some or all of the following physical characteristics: microgenia (an abnormally small chin), an unusually round face, macroglossia (protruding or oversized tongue), an almond shape to the eyes caused by an epicanthic fold of the eyelid, upslanting palpebral fissures (the separation between the upper and lower eyelids), shorter limbs, a single transverse palmar crease (a single instead of a double crease across one or both palms), poor muscle tone, and a larger than normal space between the big and second toes. Health concerns for individuals with Down syndrome include a higher risk for congenital heart defects, gastroesophageal reflux disease, recurrent ear infections that may lead to hearing loss, obstructive sleep apnea, thyroid dysfunctions, and obesity. Early childhood intervention, screening for common problems, medical treatment where indicated, a conducive family environment, and vocational training can improve the overall development of children with Down syndrome. Education and proper care will improve quality of life significantly, despite genetic limitations Down syndrome or Down's syndrome, (also known as trisomy 21), is a chromosomal condition caused by the presence of all or part of an extra 21st chromosome. It is named after John Langdon Down, the British physician who described the syndrome in 1866. The condition was clinically described earlier in the 19th century by Jean Etienne Dominique Esquirol in 1838 and Edouard Seguin in 1844. Down syndrome was identified as a chromosome 21 trisomy by Dr. Jérôme Lejeune in 1959. Down syndrome in a fetus can be identified through chorionic villus sampling or amniocentesis during pregnancy, or in a baby at birth. Down syndrome is a chromosomal condition characterized by the presence of an extra copy of genetic material on the 21st chromosome, either in whole (trisomy 21) or part (such as due to translocations). The effects and extent of the extra copy vary greatly among people, depending on genetic history, and pure chance. The incidence of Down syndrome is estimated at 1 per 733 births, although it is statistically more common with older parents due to increased mutagenic exposures upon some older parents' reproductive cells. Other factors may also play a role. Down syndrome occurs in all human populations, and analogous conditions have been found in other species such as chimpanzees and mice. Often Down syndrome is associated with some impairment of cognitive ability and physical growth, and a particular set of facial characteristics. Individuals with Down syndrome usually have low intelligence, such as to constitute mild to moderate intellectual disability. Many children with Down syndrome who have received family support, enrichment therapies and tutoring manage to graduate from high school and college, and are able to do paid work. The average IQ of children with Down syndrome is around 50, compared to normal children with an IQ of 100. A small number have a severe to high degree of intellectual disability. Individuals with Down syndrome may have some or all of the following physical characteristics: microgenia (an abnormally small chin),an unusually round face, macroglossia(protruding or oversized tongue), an almond shape to the eyes caused by an epicanthic fold of the eyelid, upslanting palpebral fissures (the separation between the upper and lower eyelids), shorter limbs, a single transverse palmar crease (a single instead of a double crease across one or both palms), poor muscle tone, and a larger than normal space between the big and second toes. Health concerns for individuals with Down syndrome include a higher risk for congenital heart defects, gastroesophageal reflux disease, recurrent ear infections that may lead to hearing loss, obstructive sleep apnea, thyroid dysfunctions, and obesity. Early childhood intervention, screening for common problems, medical treatment where indicated, a conducive family environment, and vocational training can improve the overall development of children with Down syndrome. Education and proper care will improve quality of life significantly, despite genetic limitations edward syndrome:

cB_I/ Edwards syndrome (also known as Trisomy 18 (T18) or Trisomy E) is a genetic disorder caused by the presence of all or part of an extra 18th chromosome. It is named after John H. Edwards, who first described the syndrome in 1960. It is the second most common autosomal trisomy, after Down syndrome, that carries to term. Edwards syndrome is caused by the presence of three – as opposed to two – copies of chromosome 18 in a fetus's or infant's cells. Edwards' syndrome occurs in around one in 6,000 live births and around 80 per cent of those affected are female. The majority of fetuses with the syndrome die before birth. The incidence increases as the mother's age increases. The syndrome has a very low rate of survival, resulting from heart abnormalities, kidney malformations, and other internal organ disorders. Edwards syndrome (also known as Trisomy 18 (T18) or Trisomy E) is a genetic disorder caused by the presence of all or part of an extra 18th chromosome. It is named after John H. Edwards, who first described the syndrome in 1960. It is the second most common autosomal trisomy, after Down syndrome, that carries to term. Edwards syndrome is caused by the presence of three – as opposed to two – copies of chromosome 18 in a fetus's or infant's cells. Edwards' syndrome occurs in around one in 6,000 live births and around 80 per cent of those affected are female. The majority of fetuses with the syndrome die before birth. The incidence increases as the mother's age increases. The syndrome has a very low rate of survival, resulting from heart abnormalities, kidney malformations, and other internal organ disorders. patau syndrome:

Patau syndrome, also known as trisomy 13 and trisomy D, is a chromosomal abnormality, a syndrome in which a patient has an additional chromosome 13 due to a nondisjunction of chromosomes during meiosis. Some are caused by Robertsonian translocations. The extra chromosome 13 disrupts the normal course of development, causing heart and kidney defects, amongst other features characteristic of Patau syndrome.[vague] Like all nondisjunction conditions (such as Down syndrome and Edwards syndrome), the risk of this syndrome in the offspring increases with maternal age at pregnancy, with about 31 years being the average. Patau syndrome affects somewhere between 1 in 10,000 and 1 in 21,700 live births. Patau syndrome, also known as trisomy 13 and trisomy D, is a chromosomal abnormality, a syndrome in which a patient has an additional chromosome 13 due to a nondisjunction of chromosomes during meiosis. Some are caused by Robertsonian translocations. The extra chromosome 13 disrupts the normal course of development, causing heart and kidney defects, amongst other features characteristic of Patau syndrome.[vague] Like all nondisjunction conditions (such as Down syndrome and Edwards syndrome), the risk of this syndrome in the offspring increases with maternal age at pregnancy, with about 31 years being the average. Patau syndrome affects somewhere between 1 in 10,000 and 1 in 21,700 live births.[2] albinism syndrome:

Albinism–deafness syndrome (also known as "Woolf syndrome," and "Ziprkowski–Margolis syndrome") is a condition characterized by congenital neural deafness and a severe or extreme piebald-like phenotype with extensive areas of hypopigmentation. A locus at Xq26.3-q27.I has been suggested. It has been suggested that it is a form of Waardenburg syndrome type II.














                                                                            :CONCLUSION:

               THERE I CONCLUDE I MUST THANK GOD FOR MAKING ME NORMAL
                AND  I REALLY THANKS THAT I AM NOT ONE OF THEM SUFFERING 
                 THAT DISEASE OR SYDROME  AND I MUST PRAY FOR ALL OF THEM 
                 THAT THEY WILL BE OKEY.,.,.,.  AND I LEARNED THAT MUTATION IS 
                 ACCIDENTALLY PROCIDURE OF DNA...,.,,.,

project in araling panlipunan

Corazon Cojoangco Aquino

Corazon Cojoangco Aquino (born 1933) was the first woman to run for the office of the president of the Republic of the Philippines. The results of the 1986 election were so fraudulent that both Aquino and her opponent, the incumbent, Ferdinand Marcos declared victory. As a result of the election, the Filipino people rose in protest and Marcos was forced to flee the country and Aquino assumed the office of president. Corazon Cojoangco Aquino was born on January 25, 1933, the sixth of eight children born to Jose Cojoangco of Tarlac, a prosperous province 65 miles northwest of Manila, the Philippines capital. The Cojoangcos were memberMga nagawa ni corazon tumakbo siya bilang presidente at nanalo siya ang ginawa niya sa pilipinas ay pinatakbo niya ng maayos ang bansa at sa huli ay natapos narin ang pahihirap niya AUGUST 1,2009 siya namatay at ang anak niya na si kris aquino ay hindi niya maipaliwanag ang nararamdaman niya sa sarili nang namatay ang kanyang ina si CORAZON AQUINO ay matagal na may colon cancer sa baga. s of a wealthy landowning family prominent in politics.Protecting the countryside was another of Aquino's goals. She planned to accomplish this by disarming the private armies that roamed the rural areas and establish industries there. Aquino said she would revitalize the sugar industry by breaking the monopoly. She acknowledged the special relationship with the United States but emphasized that her concern was with the Filipinos, not the Americans. Aquino knew her popularity would wane and that her leadership would be harshly criticized. At least seven coups were directed at her government during her tenure as president, many times by former allies who had helped her come to power. Besides dealing with factious parties both within her cabinet and in the nation, Aquino had to contend with natural disasters and frequent power failures. In 1991, a constitutional amendment was passed by referendum which enabled Aquino to remain president until June 30, 1992. Her successor was Fidel Ramos, her former secretary of defense and Marcos' former deputy chief of staff of the armed forces. Ramos, who assisted Aquino in fending off the coup attempts, has continued to support Aquino's democratic ideals. Aquino has still retained her popularity with the Filipino people and works for reform by participating in cooperatives and non-governmental organizations in the Philippines. Pandita Ramabai

Pandita Ramabai (23 April 1858, Maharashtra- 5 April 1922) was an eminent Indian Christian social reformer and activist. She was a poet, a scholar, and a champion of improvement in the plight of Indian women. As a social reformer, she championed the cause of emancipation of Indian women. A widely traveled lady, she visited most parts of India, and even went to England (1883) and the U.S. (1886-88). She wrote a many books including her most well known work titled The High Caste Hindu Woman,which showed the darkest of subject matter relating to the life of Hindu women, including child brides and the treatment they receive by the government. She had a strong view of what should be accomplished so women would be able to have more freedom, including protection of widowed women and child brides and she was also against the practice of suttee. Pandita Ramabai was born into an intellectual Brahmin family. Her father believed that women should have an education and against traditional Hindu social structure he taught Ramabai as well as his second wife, Ramambai’s mother [[Puranas|Puranic] and how to read and write Sanskrit. As well as how to interpret vedic texts. She was raised by her father Anant Shastri Dongre and her mother Lakshmibai. Her father was a scholar of Sanskrit, and her mother was educated as well. Annie Basant came to India in 1893, by which time she had worked with Charles Bradlaugh on the National Reformer,become a critic of British colonialism, and joined the Theosophical Society. In India she lived at Adyar in Madras, which was the headquarters of the Theosophical Society. She supported Sanatan Dharam movement advocating that Hindu children be taught Sanskrit and learn Aryan �simplicity and spirituality�, but despite this revivalist strain she believed in the equal rights of men and women on the equal but complementary basis. Initially she felt that India needed to educate herself before she was capable of self-government, but by the outbreak of the First World War she was campaigning for self- government. In 1914 she joined Congress and in 1916 was one of the founders of Home Rule League. In June 1917 she was arrested under Defense f India Act. Her arrest was followed by a series of protest meetings all over the country and abroad In 1924 she lead a deputation of home rulers to England to demand Dominion Home a Rule for India. In 1925 drafted a Bill on a constitution for self-government which was approved by Gandhi and supported by the Labor Party but never enacted. R.K. Narayan (b. 1907), a south Indian novelist who created the fictional town of Malgudi, has encompassed a broad range of women within his work. In his The Dark Room (1938), Savitri, the heroine, cannot escape from a stifling family situation because of her lack of economic independence and self-confidence. Daisy, the heroine of his last novel, The Painter of Signs (1976), is a government birth-control worker who refuses both ties to natal family and marriage proposals. Anita Desai (b.1937) has focused on the lives of urban women and has etched the divergent paths of two sisters in Clear Light of Day (1980). Memoirs, such as those by Kamaladevi Chattopadhyay (1986), Gayatri Devi of Jaipur (1976), Vijayalakshmi Pandit (1979), and Begum Shaista S. Ikramullah (1969) are important expressions of their authors� autonomy. The Nectar in the Sieve (1954) by Kamale Markandaya is the most readily available novel about peasant women, but perhaps more authentic accounts of the lives of such women are in anthropological studies,, such as Behind Mud Walls (1989), in which William and Charlotte Wiser have studied the same north Indian village over four decades from 1930 to 1970. All of these novels and memoirs were written originally in English. Although there are many Indian women writing in Indian languages, regrettably few of their novels, short stories, and poems have been translated into English or these translations are not easily available in North America. Four notable exceptions are the Longman Anthology of World Literature by Women (Arkin and Shollar 1989) that includes selections by twelve Indian women; a collection of Bengali short stories by Mahasweta Devi that Kalpana Bardhan has translated (1990); Truth Tales, edited by a collective in Delhi (1990); and a massive two-volume anthology by Susie Tharu and K. Lalita of the writings of Indian women from 600 B.C. to the present Sarojini Naidu

Sarojinidevi was a great patriot, politician, orator and administrator, of all the famous women of India, Mrs. Sarojinidevi Naidu's name is at the top. Not only that, but she was truly one of the jewels of the world. Being one of the most famous heroines of the 20th century, her birthday is celebrated as "Women's Day" She was born on February 13, 1879 in Hyderabad. Her father, Dr. Aghornath Chattopadhyaya, was the founder of Nizam College of Hyderabad and a scientist. Her mother, Mrs. Varasundari, was a Bengali poetess. Sarojinidevi inherited qualities from both her father and mother. During her stay in England, Sarojini met Dr. Govind Naidu from southern India. After finishing her studies at the age of 19, she got married to him during the time when inter-caste marriages were not allowed. Her father was a progressive thinking person, and he did not care what others said. Her marriage was a very happy one. Her major contribution was also in the field of poetry. Her poetry had beautiful words that could also be sung. Soon she got recognition as the "Bul Bule Hind" when her collection of poems was published in 1905 under the title Golden Threshold. After that, she published two other collections of poems--The Bird of Time and The Broken Wings. In 1918, Feast of Youth was published. Later, The Magic Tree, The Wizard Mask and A Treasury of Poems were published. Mahashree Arvind, Rabindranath Tagore and Jawaharlal Nehru were among the thousands of admirers of her work. Her poems had English words, but an Indian soul. One day she met Shree Gopal Krishna Gokhale. He said to her to use her poetry and her beautiful words to rejuvenate the spirit of Independence in the hearts of villagers. He asked her to use her talent to free Mother India. Then in 1916, she met Mahatma Gandhi, and she totally directed her energy to the fight for freedom. She would roam around the country like a general of the army and pour enthusiasm among the hearts of Indians. The independence of India became the heart and soul of her work. She was responsible for awakening the women of India. She brought them out of the kitchen. She traveled from state to state, city after city and asked for the rights of the women. She re-established self-esteem within the women of India. In 1925, she chaired the summit of Congress in Kanpur. In 1928, she came to the USA with the message of the non-violence movement from Gandhiji. When in 1930, Gandhiji was arrested for a protest, she took the helms of his movement. In 1931, she participated in the Round Table Summit, along with Gandhiji and Pundit Malaviyaji. In 1942, she was arrested during the "Quit India" protest and stayed in jail for 21 months with Gandhiji. After independence she became the Governor of Uttar Pradesh. She was the first woman governor in India. She died on March 2, 1949. Ichikawa Fusae (1893–1981)

Japanese suffragist, feminist, and politician, who was one of the most outstanding women in 20th-century Japan. Name variations: Ichikawa Fusaye. Pronunciation: ITCH-EE-ka-wa FOO-sa-ae. Born Ichikawa Fusae on May 15, 1893, in Asahi Village, Aichi Prefecture, Japan; died in Tokyo, Japan, in 1981; daughter of Ichikawa Fujikurō (a farmer) and Ichikawa Tatsu; attended public elementary and higher elementary schools, briefly attended Joshi Gakuin (Girls' Academy) in Tokyo, and graduated from Aichi Prefectural Women's Normal School in 1913; never married; no children. Publications: (in Japanese) Ichikawa Fusawa no jiden—senzen hen (The Autobiography of Ichikawa Fusae—The Prewar Period, 1974); Watakushi no fujin undō (My Women's Movement, 1972); Watakushi no seiji shōron (My Views of Politics, 1972); Sengo fujikai no dōkō (Trends of Women's Circles in the Postwar Period, 1969). During Ichikawa Fusae's almost 90 years, the status of Japanese women changed dramatically; women progressed from being subordinate to men, in both the private and public sphere, to being their legal equal, and she was one of those most responsible for this change. Remarkably, despite being a militant feminist, at the time of her death in 1981 Ichikawa Fusae was perhaps the most respected politician in Japan.Born to a farm family at the end of the 19th century, Ichikawa's childhood reflected both the weight of traditions which had oppressed Japanese women and the opportunities which modernization afforded them. As the head of his family, Ichikawa Fujikurō faced no censure for beating his wife; Fusae recalled seeing her mother Ichikawa Tatsuwhimpering in a corner, unable to defend herself against his blows. But her father was progressive on the issue of education, schooling his daughters, as well as his sons. For this, he tolerated the ridicule of his fellow villagers. Fusae claimed that she was raised to be "bold or aggressive," to ignore conventional propriety—a trait she would exhibit throughout her life. After attending elementary school, she was briefly enrolled at one of the most progressive girls' schools in Tokyo, Joshi Gakuin (Girls' Academy), whose director, Yajima Kajiko, was an outspoken advocate of women's rights. Between 1909 and 1913, … Aung San Suu Kyi

1942: September 6. Marriage of Aung San, commander of the Burma Independence Army, and Ma Khin Kyi (becoming Daw Khin Kyi), senior nurse of Rangoon General Hospital, where he had recovered from the rigours of the march into Burma. 1945: June 19. Aung San Suu Kyi born in Rangoon, third child in family. "Aung San" for father, "Kyi" for mother, "Suu" for grandmother, also day of week of birth. Favourite brother is to drown tragically at an early age. The older brother, will settle in San Diego, California, becoming United States citizen. 1947: July 19. General Aung San assassinated. Suu Kyi is two years old. Daw Khin Kyi becomes a prominent public figure, heading social planning and social policy bodies. 1948: January 4. The Independent Union of Burma is established. 1960: Daw Khin Kyi appointed Burma's ambassador to India. Suu Kyi accompanies mother to New Delhi. 1960-64: Suu Kyi at high school and Lady Shri Ram College in New Delhi. 1964-67: Oxford University, B.A. in philosophy, politics and economics at St. Hugh's College (elected Honorary Fellow, 1990). British "parents" are Lord Gore-Booth, former British ambassador to Burma and High Commissioner in India, and his wife, at whose home Suu Kyi meets Michael Aris, student of Tibetan civilisation. 1969-71: She goes to New York for graduate study, staying with family friend Ma Than E, staff member at the United Nations, where U. Thant of Burma is Secretary-General. Postponing studies, Suu Kyi joins U.N. secretariat as Assistant Secretary, Advisory Committee on Administrative and Budgetary Questions. Evenings and weekends volunteers at hospital, helping indigent patients in programs of reading and companionship. 1972: January 1. Marries Michael Aris, joins him in Himalayan kingdom of Bhutan, where he tutors royal family and heads Translation Department. She becomes Research Officer in the Royal Ministry of Foreign Affairs. 1973: They return to England for birth of Alexander in London. 1974: Michael assumes appointment in Tibetan and Himalayan studies at Oxford University. 1977: Birth of second son, Kim at Oxford. While raising her children, Suu Kyi begins writing, researches for biography of father, and assists Michael in Himalayan studies. 1984: Publishes Aung San in Leaders of Asia series of University of Queensland Press. (See Freedom from Fear, pp. 3-38.) 1985: For juvenile readers publishes Let's Visit Burma (see Freedom from Fear, pp. 39-81), also books on Nepal and Bhutan in same series for Burke Publishing Company, London. 1985-86: Visiting Scholar, Center of Southeast Asian Studies, Kyoto University, researching father's time in Japan. Kim with her, Alexander with Michael, who has fellowship at Indian Institute of Advanced Studies at Simla in northern India. 1986: On annual visit to grandmother in Rangoon, Alexander and Kim take part in traditional Buddhist ceremony of initiation into monkhood. 1987: With fellowship at Indian Institute Suu Kyi, with Kim, joins Michael and Alexander in Simla. Travels to London when mother is there for cataract surgery. Publishes "Socio-Political Currents in Burmese Literature, 1910-1940" in journal of Tokyo University. (See Freedom from Fear, pp. 140-164.) September. Family returns to Oxford. Suu Kyi enrolls at London School of Oriental and African Studies to work on advanced degree. 1988: March 31. Informed by telephone of mother's severe stroke, she takes plane next day to Rangoon to help care for Daw Khin Kyi at hospital, then moves her to family home on University Avenue next to Inya Lake in Rangoon. July 23. Resignation of General Ne Win, since 1962 military dictator of Burma. Popular demonstrations of protest continuing. August 8. Mass uprising throughout country. Violent suppression by military kills thousands. August 15. Suu Kyi, in first political action, sends open letter to government, asking for formation of independent consultative committee to prepare multi-party elections. August 26. In first public speech, she addresses several hundred thousand people outside Shwedagon Pagoda, calling for democratic government. Michael and her two sons are there. September 18. Military establishes State Law and Order Restoration Council (SLORC). Political gatherings of more than four persons banned. Arrests and sentencing without trial reaffirmed. Parliamentary elections to be held, but in expectation that multiplicity of parties will prevent clear result. September 24. National League for Democracy (NLD) formed, with Suu Kyi general-secretary. Policy of non-violence and civil disobedience. October-December. Defying ban, Suu Kyi makes speech-making tour throughout country to large audiences. December 27. Daw Khin Kyi dies at age of seventy-six. 1989: January 2. Funeral of Daw Khin Kyi. Huge funeral procession. Suu Kyi vows that as her father and mother had served the people of Burma, so too would she, even unto death. January-July. Suu Kyi continues campaign despite harassment, arrests and killings by soldiers. February 17. Suu Kyi prohibited from standing for election. April 5. Incident in Irawaddy Delta when Suu Kyi courageously walks toward rifles soldiers are aiming at her. July 20. Suu Kyi placed under house arrest, without charge or trial. Sons already with her. Michael flies to Rangoon, finds her on third day of hunger strike, asking to be sent to prison to join students arrested at her home. Ends strike when good treatment of students is promised. 1990: May 27. Despite detention of Suu Kyi, NLD wins election with 82% of parliamentary seats. SLORC refuses to recognise results. October 12. Suu Kyi granted 1990 Rafto Human Rights Prize. 1991: July 10. European Parliament awards Suu Kyi Sakharov human rights prize. October 14. Norwegian Nobel Committee announces Suu Kyi is winner of 1991 Peace Prize. 1991: December. Freedom from Fear published by Penguin in New York, England, Canada, Australia, New Zealand. Also in Norwegian, French, Spanish translations. December 10. Alexander and Kim accept prize for mother in Oslo ceremony. Suu Kyi remains in detention, having rejected offer to free her if she will leave Burma and withdraw from politics. Worldwide appeal growing for her release. 1992: Suu Kyi announces that she will use $1.3 million prize money to establish health and education trust for Burmese people. 1993: Group of Nobel Peace Laureates, denied entry to Burma, visit Burmese refugees on Thailand border, call for Suu Kyi's release, Their appeal later repeated at UN Commission for Human Rights in Geneva. 1994: February. First non-family visitors to Suu Kyi: UN representative, U.S. congressman, New York Times reporter. September-October. SLORC leaders meet with Suu Kyi, who still asks for a public dialogue. 1995: July 10. SLORC releases Suu Kyi from house arrest after six years of detention. Freedom from Fear and Other Writings. Edited with introduction by Michael Aris. 2nd ed., revised. New York and London: Penguin, 1995. (Includes essays by friends and scholars.) Voice of Hope: Conversations. London: Penguin, 1997 and New York City: Seven Stories Press, 1997 (Conversations beginning in November 1995 with Alan Clements, the founder of the Burma Project in California who helped with the script for the film based on her life, “Beyond Rangoon”.) Other Sources “Aung San Suu Kyi”, in Current Biography, February 1992. Clements, Alan and Leslie Kean. Burma’s Revolution of the Spirit: The Struggle for Democratic Freedom and Dignity. New York: Aperture, 1994. (Many colour photographs with text, Includes essay by Aung San Suu Kyi.) Clements, Alan. Burma: The Next Killing Fields. Tucson, Arizona; Odonian Press, 1992. (With a foreword by the Dalai Lama.) Lintner, Bertil. Burma in Revolt: Opium and Insurgency since 1948. Boulder. Colorado: Westview, 1994. (By a well-informed Swedish journalist.) Lintner, Bertil. Outrage: Burma’s Struggle for Democracy. 2nd ed., Edinburgh: Kiscadale, 1995. Mirante, Edith T. Burmese Looking Glass. A Human Rights Adventure and a Jungle Revolution. New York: Grove, 1993. Smith, Martin J. Burma: Intrangency and the Politics of Ethnicity. London: Zed Books, 1991. (A detailed and well-organised account by a journalist of the violent conflict between the military government and the many minorities.) Victor, Barbara. The Lady: Aung San Suu Kyi: Nobel Laureate and Burma’s Prisoner. Boston and London: Faber & Faber, 1998. (A sympathetic account by a wellpublished author and journalist, whose research in Burma included interviews with government leaders.)

project in a.c.t

Genetic disorders Genetic disorders are conditions that have some origin in an individual's genetic make-up. Many of these disorders are inherited and are governed by the same genetic rules that determine dimples and red hair. However, some genetic disorders—such as Down syndrome, characterized by heart malformation, poor muscle tone, and a flattened face—result from a spontaneous mutation (gene change) that takes place during embryonic (earliest life) development. Genetic disorders can be classified according to the way in which they develop. If the disorder is transmitted by genes inherited from only one parent, it is said to be an autosomal dominant disorder. The term autosome applies to any of the 22 chromosomes that are identical in human males and females. (Chromosomes are structures that organize genetic information in the nucleus of cells.) By contrast, disorders that can be inherited only by the transmission of genes from both parents is called an autosomal recessive disorder. Other genetic disorders are associated with the X (female) or Y (male) chromosome and are called sex-linked disorders because the X and Y chromosomes are related to sexual characteristics in humans. Finally, the development of some genetic disorders involves environmental factors, factors present outside the organism itself. Such disorders are known as multifactorial genetic disorders. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o3ENydbQ Principles of genetic inheritance Genetic information in humans is stored in units known as genes located on large complex molecules called chromosomes. A vast range of human characteristics, from eye and hair color to musical and literary talents, are controlled by genes. To say that a person has red hair color, for example, is simply to say that that person's body contains genes that tell hair cells how to make red hair. Reproduction in humans occurs when a sperm cell from a male penetrates and fertilizes an egg cell from a female. The fertilized egg cell, called a zygote, contains genes from both parents. For example, the zygote will contain two genes that control hair color, one gene from the mother and one gene from the father. In some cases, both genes carry the same message. For example, the zygote might contain two genes that act as a kind of code that tells a cell to make red hair, one from each parent. In that case, the child will be born with red hair. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o3Ec9cAi Words to Know Chromosomes: Structures that organize genetic information in the nucleus of cells. Dominant trait: A trait that can manifest (be expressed) when inherited from one parent. Gene: A section of a chromosome that carries instructions for the formation, functioning, and transmission of specific traits from one generation to another. Multifactorial trait: A trait that results from both genetic and environmental influences. Proteins: Large molecules that are essential to the structure and functioning of all living cells. Recessive trait: A trait that is expressed in offspring only when identical genes for the trait are inherited from both parents. Sex-linked disorder: A disorder that generally affects only one sex (male or female). In other cases, two genes may carry different messages. The zygote might, for instance, carry a gene for red hair from the mother and for brown hair from the father. In such cases, one gene is dominant and the other recessive. As these terms suggest, one gene will "win out" over the other and determine the offspring's hair color. In this example, the gene for brown hair is dominant over the gene for red hair, and the offspring will have brown hair. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o3EiTO00 "5 EXAMPLES OF GENETICS DISORDER Dominant genetic disorders If one parent has an autosomal dominant disorder, then offspring have a 50 percent chance of inheriting that disease. Approximately 2,000 autosomal dominant disorders (ADDs) have been identified. These disorders have effects that range from inconvenience to death. ADDs include Huntington's disorder, polydactyly (extra toes or fingers), Marfan's syndrome (extra long limbs), achondroplasia (a type of dwarfism), some forms of glaucoma (a vision disorder), and hypercholesterolemia (high blood cholesterol). ADDs may occur early or late in life. People with ADDs that are diagnosed at older ages are faced with very special problems. They may already have had children of their own and transmitted the genetic trait that caused their disorder to their offspring. Huntington's disease (also known as Huntington's chorea) is an example of an ADD that is typically diagnosed relatively late in life. The Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o34xxccV

A young girl with the genetic disorder Down syndrome. (Reproduced by permission of Photo Researchers, Inc. ) disorder is characterized by progressive involuntary, rapid, jerky motions and mental deterioration. It usually appears in affected individuals between the ages of 30 and 50, and leads to dementia and eventual death in about 15 years. Marfan's syndrome, also called arachnodactyly, is an ADD characterized by long, thin arms, legs, and fingers. People with Marfan's syndrome also tend to be stoop-shouldered and have a bluish tint to their eyeballs. In addition, these individuals have a high incidence of eye and heart problems. Abraham Lincoln is believed to have had Marfan's syndrome. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o35Qp2YR Recessive genetic disorders Recessive genetic disorders (RGD) are caused when both parents supply a recessive gene to their offspring. The probability of such an event's occurring is 25 percent each time the parents conceive. About 1,000 confirmed RGDs exist. Some of the better known examples of the condition include cystic fibrosis, sickle-cell anemia, Tay-Sachs disease, galactosemia, phenylketonuria (PKU), adenosine deaminase deficiency, growth hormone deficiency, Werner's syndrome (juvenile muscular dystrophy), albinism (lack of skin pigment), and autism. Some RGDs tend to affect people of one particular ethnic background at a higher rate than the rest of the population. Three such RGDs are cystic fibrosis, sickle-cell anemia, and Tay-Sachs disease. Cystic fibrosis is one of the most common autosomal recessive diseases in Caucasian children in the United States. About 5 percent of Caucasians carry this recessive gene. Cystic fibrosis is characterized by excessive secretion of an unusually thick mucus that clogs respiratory ducts and collects in lungs and other body areas. Cystic fibrosis patients usually die before the age of 20, although some individuals live to the age of 30. Sickle-cell anemia occurs with an unusually high incidence among the world's black and Hispanic populations. However, some cases also occur in Italian, Greek, Arabian, Maltese, southern Asian, and Turkish people. About 1 in 12 blacks carry the gene for this disorder. Sickle-cell anemia is caused by mutations in the genes responsible for the production of hemoglobin. (Hemoglobin is the compound that carries oxygen in red blood cells to tissues and organs throughout the body.) Sickle-cell anemia patients have red blood cells that live only a fraction of the normal life span of 120 days. The abnormal blood cells have a sickled appearance, which led to the disease's name. Sickle-cell patients also die early, before the age of 30. The Tay-Sachs gene is carried by 1 in 30 Ashkenazi Jews. Children born with Tay-Sachs disorder seem normal for the first 5 months of their lives. But afterwards, they begin to express symptoms of the disorder. Eventually, the condition leads to blindness and death before the age of four. Galactosemia and PKU are examples of metabolic RGDs. A metabolic RGD is one in which a person's body is unable to carry out functions that are normal and essential to the body. For example, people with galactosemia lack an enzyme (chemical) needed to metabolize (break down) galactose, a sugar found in milk. If people with galactosemia do not avoid normal milk, mental retardation will eventually develop. People with PKU have a similar problem. They lack an enzyme needed to convert the amino acid phenylalanine to the amino acid tyrosine. The build-up of phenylalanine in the body leads to severe mental retardation. Adenosine deaminase deficiency is one of few "curable" genetic diseases. It is caused by a mutation in a single gene essential to normal development of the immune system. Bone marrow transplants have been found to be of some value to patients. In addition, gene therapy has been successful at replacing these patients' defective gene with a copy of a correct gene that enables their immune system to function effectively. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o35sGKvv

Color Blindness

Is the traffic light red or green? Most humans have the ability to distinguish the color we call red from the color we call green. But some people cannot. Such people are said to be color-blind. Color blindness is a defect in vision that makes it difficult or impossible for a person to distinguish between or among certain colors. Color-blindness is usually passed on genetically, and is more common in men than in women. About 6 percent of all men and roughly one-tenth of that many women inherit the condition. Individuals also can acquire the condition through various eye diseases. There is no treatment for color blindness. The most common form of color-blindness involves the inability to distinguish reds from greens. A less common condition involves the inability to distinguish green from yellow. Color blindness is caused by a lack of pigment in the retina of the eye. Normally, the retina contains molecules capable of detecting every color in the spectrum. However, if some of these molecules are not present, the various colors in the spectrum can not be distinguished from each other, and the person is color-blind. Color blindness is a sex-linked characteristic. The gene involved in the disorder occurs only on the X chromosome, which is passed to the child by the mother. The Y chromosome, which is passed to the child by the father, does not carry the defective gene. As a result, children inherit color blindness only from their mothers. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o36a2zIw Sex-linked genetic disorders

Sex-linked genetic disorders (XLGDs) can be either dominant or recessive. Dominant XLGDs affect females, are usually fatal, and cause severe disorders in males who survive. A high percentage of male embryos with dominant XLGD spontaneously abort early in a pregnancy. Dominant XLGD's include conditions such as Albright's hereditary osteodystrophy (seizures, mental retardation, stunted growth), Goltz's syndrome (mental retardation), cylindromatosis (deafness and upper body tumors), oral-facial-digital syndrome (no teeth, cleft tongue, some mental retardation), and incontinentia pigmenti (abnormal swirled skin pigmentation). Recessive XLGDs are passed to sons through their mothers. Major XLGDs include severe combined immune deficiency syndrome (SCID), color blindness, hemophilia, Duchenne's muscular dystrophy (DMD), some spinal ataxias, and Lesch-Nyhan syndrome. Roughly one-third of these XLGDs result from a spontaneous mutation. Of these disorders, color blindness is the least harmful. Hemophilia is an example of a serious XLGD. This disorder is caused by the absence of a protein responsible for the clotting of blood. Lacking this protein, a person with hemophilia may easily bleed to death from simple cuts and injuries that would be of little danger to the average person. Hemophilia A is the most severe form of this disease, and is characterized by extreme bleeding. It affects males primarily, although it has been known to occur in females. The disorder has often been associated with royalty. England's Queen Victoria was a carrier whose descendants became rulers in several European countries. Other usually fatal XLGDs affect the immune, muscular, and nervous systems. SCID, for example, is a disorder affecting the immune system. It is characterized by a very poor ability to combat infection. One way to treat patients with SCID is to completely enclose them in a large plastic bubble that protects them from germs present in the air. The only known cure for SCID involves a bone marrow transplant from a close relative. DMD afflicts young boys and is apparent by age three or four. It is characterized by wasting leg and pelvic muscles. Patients with DMD are usually wheelchair-bound by the age of 12, and die before the age of 20, often as the result of heart problems. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o3739Ywf Multifactorial genetic disorders

cientists often find it difficult to determine the relative role of heredity and environment in certain medical disorders. One way to answer this question is with statistical and twin studies. Identical and fraternal twins who have been raised in different and identical homes are evaluated for these MFGDs. If fraternal twins have a higher than normal incidence of a disorder and identical twins show an even higher rate of the disorder, then genetic inheritance is believed to contribute to development of the disorder. Among the most likely candidates for multifactorial genetic disorders are certain medical conditions associated with diet and metabolism, such as obesity, diabetes, alcoholism, rickets, and high blood pressure; some infectious diseases, such as measles, scarlet fever, and tuberculosis; schizophrenia and some other psychological illnesses; club foot and cleft lip; and various forms of cancer. The tendency of some people to be more susceptible to a particular MFGD and not another is characteristic of human genetics. All healthy humans have a similar body form with very similar physiological functions. Still, it is easy to see tremendous human diversity that results from a diverse gene pool. This diversity explains why certain groups of people with similar kinds of genes are more prone to some disorders, whereas others have resistance to the same disorders. This diversity protects the human race from being wiped out by a single kind of medical problem. Read more: Genetic Disorders - humans, examples, body, life, type, chemical, characteristics, form, system, oxygen, air, cells, effects, cause, change, Principles of genetic inheritance http://www.scienceclarified.com/Ga-He/Genetic-Disorders.html#ixzz1o37WltzW

project in aral pan....

"Corazon Cojoangco Aquino" Corazon Cojoangco Aquino (born 1933) was the first woman to run for the office of the president of the Republic of the Philippines. The results of the 1986 election were so fraudulent that both Aquino and her opponent, the incumbent, Ferdinand Marcos declared victory. As a result of the election, the Filipino people rose in protest and Marcos was forced to flee the country and Aquino assumed the office of president. Corazon Cojoangco Aquino was born on January 25, 1933, the sixth of eight children born to Jose Cojoangco of Tarlac, a prosperous province 65 miles northwest of Manila, the Philippines capital. The Cojoangcos were memberMga nagawa ni corazon tumakbo siya bilang presidente at nanalo siya ang ginawa niya sa pilipinas ay pinatakbo niya ng maayos ang bansa at sa huli ay natapos narin ang pahihirap niya AUGUST 1,2009 siya namatay at ang anak niya na si kris aquino ay hindi niya maipaliwanag ang nararamdaman niya sa sarili nang namatay ang kanyang ina si CORAZON AQUINO ay matagal na may colon cancer sa baga. s of a wealthy landowning family prominent in politics.Protecting the countryside was another of Aquino's goals. She planned to accomplish this by disarming the private armies that roamed the rural areas and establish industries there. Aquino said she would revitalize the sugar industry by breaking the monopoly. She acknowledged the special relationship with the United States but emphasized that her concern was with the Filipinos, not the Americans. Aquino knew her popularity would wane and that her leadership would be harshly criticized. At least seven coups were directed at her government during her tenure as president, many times by former allies who had helped her come to power. Besides dealing with factious parties both within her cabinet and in the nation, Aquino had to contend with natural disasters and frequent power failures. In 1991, a constitutional amendment was passed by referendum which enabled Aquino to remain president until June 30, 1992. Her successor was Fidel Ramos, her former secretary of defense and Marcos' former deputy chief of staff of the armed forces. Ramos, who assisted Aquino in fending off the coup attempts, has continued to support Aquino's democratic ideals. Aquino has still retained her popularity with the Filipino people and works for reform by participating in cooperatives and non-governmental organizations in the Philippines. Pandita Ramabai Pandita Ramabai (23 April 1858, Maharashtra- 5 April 1922) was an eminent Indian Christian social reformer and activist. She was a poet, a scholar, and a champion of improvement in the plight of Indian women. As a social reformer, she championed the cause of emancipation of Indian women. A widely traveled lady, she visited most parts of India, and even went to England (1883) and the U.S. (1886-88). She wrote a many books including her most well known work titled The High Caste Hindu Woman,which showed the darkest of subject matter relating to the life of Hindu women, including child brides and the treatment they receive by the government. She had a strong view of what should be accomplished so women would be able to have more freedom, including protection of widowed women and child brides and she was also against the practice of suttee. Pandita Ramabai was born into an intellectual Brahmin family. Her father believed that women should have an education and against traditional Hindu social structure he taught Ramabai as well as his second wife, Ramambai’s mother [[Puranas|Puranic] and how to read and write Sanskrit. As well as how to interpret vedic texts. She was raised by her father Anant Shastri Dongre and her mother Lakshmibai. Her father was a scholar of Sanskrit, and her mother was educated as well. Annie Basant came to India in 1893, by which time she had worked with Charles Bradlaugh on the National Reformer,become a critic of British colonialism, and joined the Theosophical Society. In India she lived at Adyar in Madras, which was the headquarters of the Theosophical Society. She supported Sanatan Dharam movement advocating that Hindu children be taught Sanskrit and learn Aryan �simplicity and spirituality�, but despite this revivalist strain she believed in the equal rights of men and women on the equal but complementary basis. Initially she felt that India needed to educate herself before she was capable of self-government, but by the outbreak of the First World War she was campaigning for self- government. In 1914 she joined Congress and in 1916 was one of the founders of Home Rule League. In June 1917 she was arrested under Defense f India Act. Her arrest was followed by a series of protest meetings all over the country and abroad In 1924 she lead a deputation of home rulers to England to demand Dominion Home a Rule for India. In 1925 drafted a Bill on a constitution for self-government which was approved by Gandhi and supported by the Labor Party but never enacted. R.K. Narayan (b. 1907), a south Indian novelist who created the fictional town of Malgudi, has encompassed a broad range of women within his work. In his The Dark Room (1938), Savitri, the heroine, cannot escape from a stifling family situation because of her lack of economic independence and self-confidence. Daisy, the heroine of his last novel, The Painter of Signs (1976), is a government birth-control worker who refuses both ties to natal family and marriage proposals. Anita Desai (b.1937) has focused on the lives of urban women and has etched the divergent paths of two sisters in Clear Light of Day (1980). Memoirs, such as those by Kamaladevi Chattopadhyay (1986), Gayatri Devi of Jaipur (1976), Vijayalakshmi Pandit (1979), and Begum Shaista S. Ikramullah (1969) are important expressions of their authors� autonomy. The Nectar in the Sieve (1954) by Kamale Markandaya is the most readily available novel about peasant women, but perhaps more authentic accounts of the lives of such women are in anthropological studies,, such as Behind Mud Walls (1989), in which William and Charlotte Wiser have studied the same north Indian village over four decades from 1930 to 1970. All of these novels and memoirs were written originally in English. Although there are many Indian women writing in Indian languages, regrettably few of their novels, short stories, and poems have been translated into English or these translations are not easily available in North America. Four notable exceptions are the Longman Anthology of World Literature by Women (Arkin and Shollar 1989) that includes selections by twelve Indian women; a collection of Bengali short stories by Mahasweta Devi that Kalpana Bardhan has translated (1990); Truth Tales, edited by a collective in Delhi (1990); and a massive two-volume anthology by Susie Tharu and K. Lalita of the writings of Indian women from 600 B.C. to the present Sarojini Naidu Sarojini Naidu Sarojinidevi was a great patriot, politician, orator and administrator, of all the famous women of India, Mrs. Sarojinidevi Naidu's name is at the top. Not only that, but she was truly one of the jewels of the world. Being one of the most famous heroines of the 20th century, her birthday is celebrated as "Women's Day" She was born on February 13, 1879 in Hyderabad. Her father, Dr. Aghornath Chattopadhyaya, was the founder of Nizam College of Hyderabad and a scientist. Her mother, Mrs. Varasundari, was a Bengali poetess. Sarojinidevi inherited qualities from both her father and mother. During her stay in England, Sarojini met Dr. Govind Naidu from southern India. After finishing her studies at the age of 19, she got married to him during the time when inter-caste marriages were not allowed. Her father was a progressive thinking person, and he did not care what others said. Her marriage was a very happy one. Her major contribution was also in the field of poetry. Her poetry had beautiful words that could also be sung. Soon she got recognition as the "Bul Bule Hind" when her collection of poems was published in 1905 under the title Golden Threshold. After that, she published two other collections of poems--The Bird of Time and The Broken Wings. In 1918, Feast of Youth was published. Later, The Magic Tree, The Wizard Mask and A Treasury of Poems were published. Mahashree Arvind, Rabindranath Tagore and Jawaharlal Nehru were among the thousands of admirers of her work. Her poems had English words, but an Indian soul. One day she met Shree Gopal Krishna Gokhale. He said to her to use her poetry and her beautiful words to rejuvenate the spirit of Independence in the hearts of villagers. He asked her to use her talent to free Mother India. Then in 1916, she met Mahatma Gandhi, and she totally directed her energy to the fight for freedom. She would roam around the country like a general of the army and pour enthusiasm among the hearts of Indians. The independence of India became the heart and soul of her work. She was responsible for awakening the women of India. She brought them out of the kitchen. She traveled from state to state, city after city and asked for the rights of the women. She re-established self-esteem within the women of India. In 1925, she chaired the summit of Congress in Kanpur. In 1928, she came to the USA with the message of the non-violence movement from Gandhiji. When in 1930, Gandhiji was arrested for a protest, she took the helms of his movement. In 1931, she participated in the Round Table Summit, along with Gandhiji and Pundit Malaviyaji. In 1942, she was arrested during the "Quit India" protest and stayed in jail for 21 months with Gandhiji. After independence she became the Governor of Uttar Pradesh. She was the first woman governor in India. She died on March 2, 1949. Ichikawa Fusae (1893–1981) Japanese suffragist, feminist, and politician, who was one of the most outstanding women in 20th-century Japan. Name variations: Ichikawa Fusaye. Pronunciation: ITCH-EE-ka-wa FOO-sa-ae. Born Ichikawa Fusae on May 15, 1893, in Asahi Village, Aichi Prefecture, Japan; died in Tokyo, Japan, in 1981; daughter of Ichikawa Fujikurō (a farmer) and Ichikawa Tatsu; attended public elementary and higher elementary schools, briefly attended Joshi Gakuin (Girls' Academy) in Tokyo, and graduated from Aichi Prefectural Women's Normal School in 1913; never married; no children. Publications: (in Japanese) Ichikawa Fusawa no jiden—senzen hen (The Autobiography of Ichikawa Fusae—The Prewar Period, 1974); Watakushi no fujin undō (My Women's Movement, 1972); Watakushi no seiji shōron (My Views of Politics, 1972); Sengo fujikai no dōkō (Trends of Women's Circles in the Postwar Period, 1969). During Ichikawa Fusae's almost 90 years, the status of Japanese women changed dramatically; women progressed from being subordinate to men, in both the private and public sphere, to being their legal equal, and she was one of those most responsible for this change. Remarkably, despite being a militant feminist, at the time of her death in 1981 Ichikawa Fusae was perhaps the most respected politician in Japan.Born to a farm family at the end of the 19th century, Ichikawa's childhood reflected both the weight of traditions which had oppressed Japanese women and the opportunities which modernization afforded them. As the head of his family, Ichikawa Fujikurō faced no censure for beating his wife; Fusae recalled seeing her mother Ichikawa Tatsuwhimpering in a corner, unable to defend herself against his blows. But her father was progressive on the issue of education, schooling his daughters, as well as his sons. For this, he tolerated the ridicule of his fellow villagers. Fusae claimed that she was raised to be "bold or aggressive," to ignore conventional propriety—a trait she would exhibit throughout her life. After attending elementary school, she was briefly enrolled at one of the most progressive girls' schools in Tokyo, Joshi Gakuin (Girls' Academy), whose director, Yajima Kajiko, was an outspoken advocate of women's rights. Between 1909 and 1913, … Aung San Suu Kyi 1942: September 6. Marriage of Aung San, commander of the Burma Independence Army, and Ma Khin Kyi (becoming Daw Khin Kyi), senior nurse of Rangoon General Hospital, where he had recovered from the rigours of the march into Burma. 1945: June 19. Aung San Suu Kyi born in Rangoon, third child in family. "Aung San" for father, "Kyi" for mother, "Suu" for grandmother, also day of week of birth. Favourite brother is to drown tragically at an early age. The older brother, will settle in San Diego, California, becoming United States citizen. 1947: July 19. General Aung San assassinated. Suu Kyi is two years old. Daw Khin Kyi becomes a prominent public figure, heading social planning and social policy bodies. 1948: January 4. The Independent Union of Burma is established. 1960: Daw Khin Kyi appointed Burma's ambassador to India. Suu Kyi accompanies mother to New Delhi. 1960-64: Suu Kyi at high school and Lady Shri Ram College in New Delhi. 1964-67: Oxford University, B.A. in philosophy, politics and economics at St. Hugh's College (elected Honorary Fellow, 1990). British "parents" are Lord Gore-Booth, former British ambassador to Burma and High Commissioner in India, and his wife, at whose home Suu Kyi meets Michael Aris, student of Tibetan civilisation. 1969-71: She goes to New York for graduate study, staying with family friend Ma Than E, staff member at the United Nations, where U. Thant of Burma is Secretary-General. Postponing studies, Suu Kyi joins U.N. secretariat as Assistant Secretary, Advisory Committee on Administrative and Budgetary Questions. Evenings and weekends volunteers at hospital, helping indigent patients in programs of reading and companionship. 1972: January 1. Marries Michael Aris, joins him in Himalayan kingdom of Bhutan, where he tutors royal family and heads Translation Department. She becomes Research Officer in the Royal Ministry of Foreign Affairs. 1973: They return to England for birth of Alexander in London. 1974: Michael assumes appointment in Tibetan and Himalayan studies at Oxford University. 1977: Birth of second son, Kim at Oxford. While raising her children, Suu Kyi begins writing, researches for biography of father, and assists Michael in Himalayan studies. 1984: Publishes Aung San in Leaders of Asia series of University of Queensland Press. (See Freedom from Fear, pp. 3-38.) 1985: For juvenile readers publishes Let's Visit Burma (see Freedom from Fear, pp. 39-81), also books on Nepal and Bhutan in same series for Burke Publishing Company, London. 1985-86: Visiting Scholar, Center of Southeast Asian Studies, Kyoto University, researching father's time in Japan. Kim with her, Alexander with Michael, who has fellowship at Indian Institute of Advanced Studies at Simla in northern India. 1986: On annual visit to grandmother in Rangoon, Alexander and Kim take part in traditional Buddhist ceremony of initiation into monkhood. 1987: With fellowship at Indian Institute Suu Kyi, with Kim, joins Michael and Alexander in Simla. Travels to London when mother is there for cataract surgery. Publishes "Socio-Political Currents in Burmese Literature, 1910-1940" in journal of Tokyo University. (See Freedom from Fear, pp. 140-164.) September. Family returns to Oxford. Suu Kyi enrolls at London School of Oriental and African Studies to work on advanced degree. 1988: March 31. Informed by telephone of mother's severe stroke, she takes plane next day to Rangoon to help care for Daw Khin Kyi at hospital, then moves her to family home on University Avenue next to Inya Lake in Rangoon. July 23. Resignation of General Ne Win, since 1962 military dictator of Burma. Popular demonstrations of protest continuing. August 8. Mass uprising throughout country. Violent suppression by military kills thousands. August 15. Suu Kyi, in first political action, sends open letter to government, asking for formation of independent consultative committee to prepare multi-party elections. August 26. In first public speech, she addresses several hundred thousand people outside Shwedagon Pagoda, calling for democratic government. Michael and her two sons are there. September 18. Military establishes State Law and Order Restoration Council (SLORC). Political gatherings of more than four persons banned. Arrests and sentencing without trial reaffirmed. Parliamentary elections to be held, but in expectation that multiplicity of parties will prevent clear result. September 24. National League for Democracy (NLD) formed, with Suu Kyi general-secretary. Policy of non-violence and civil disobedience. October-December. Defying ban, Suu Kyi makes speech-making tour throughout country to large audiences. December 27. Daw Khin Kyi dies at age of seventy-six. 1989: January 2. Funeral of Daw Khin Kyi. Huge funeral procession. Suu Kyi vows that as her father and mother had served the people of Burma, so too would she, even unto death. January-July. Suu Kyi continues campaign despite harassment, arrests and killings by soldiers. February 17. Suu Kyi prohibited from standing for election. April 5. Incident in Irawaddy Delta when Suu Kyi courageously walks toward rifles soldiers are aiming at her. July 20. Suu Kyi placed under house arrest, without charge or trial. Sons already with her. Michael flies to Rangoon, finds her on third day of hunger strike, asking to be sent to prison to join students arrested at her home. Ends strike when good treatment of students is promised. 1990: May 27. Despite detention of Suu Kyi, NLD wins election with 82% of parliamentary seats. SLORC refuses to recognise results. October 12. Suu Kyi granted 1990 Rafto Human Rights Prize. 1991: July 10. European Parliament awards Suu Kyi Sakharov human rights prize. October 14. Norwegian Nobel Committee announces Suu Kyi is winner of 1991 Peace Prize. 1991: December. Freedom from Fear published by Penguin in New York, England, Canada, Australia, New Zealand. Also in Norwegian, French, Spanish translations. December 10. Alexander and Kim accept prize for mother in Oslo ceremony. Suu Kyi remains in detention, having rejected offer to free her if she will leave Burma and withdraw from politics. Worldwide appeal growing for her release. 1992: Suu Kyi announces that she will use $1.3 million prize money to establish health and education trust for Burmese people. 1993: Group of Nobel Peace Laureates, denied entry to Burma, visit Burmese refugees on Thailand border, call for Suu Kyi's release, Their appeal later repeated at UN Commission for Human Rights in Geneva. 1994: February. First non-family visitors to Suu Kyi: UN representative, U.S. congressman, New York Times reporter. September-October. SLORC leaders meet with Suu Kyi, who still asks for a public dialogue. 1995: July 10. SLORC releases Suu Kyi from house arrest after six years of detention. Freedom from Fear and Other Writings. Edited with introduction by Michael Aris. 2nd ed., revised. New York and London: Penguin, 1995. (Includes essays by friends and scholars.) Voice of Hope: Conversations. London: Penguin, 1997 and New York City: Seven Stories Press, 1997 (Conversations beginning in November 1995 with Alan Clements, the founder of the Burma Project in California who helped with the script for the film based on her life, “Beyond Rangoon”.) Other Sources “Aung San Suu Kyi”, in Current Biography, February 1992. Clements, Alan and Leslie Kean. Burma’s Revolution of the Spirit: The Struggle for Democratic Freedom and Dignity. New York: Aperture, 1994. (Many colour photographs with text, Includes essay by Aung San Suu Kyi.) Clements, Alan. Burma: The Next Killing Fields. Tucson, Arizona; Odonian Press, 1992. (With a foreword by the Dalai Lama.) Lintner, Bertil. Burma in Revolt: Opium and Insurgency since 1948. Boulder. Colorado: Westview, 1994. (By a well-informed Swedish journalist.) Lintner, Bertil. Outrage: Burma’s Struggle for Democracy. 2nd ed., Edinburgh: Kiscadale, 1995. Mirante, Edith T. Burmese Looking Glass. A Human Rights Adventure and a Jungle Revolution. New York: Grove, 1993. Smith, Martin J. Burma: Intrangency and the Politics of Ethnicity. London: Zed Books, 1991. (A detailed and well-organised account by a journalist of the violent conflict between the military government and the many minorities.) Victor, Barbara. The Lady: Aung San Suu Kyi: Nobel Laureate and Burma’s Prisoner. Boston and London: Faber & Faber, 1998. (A sympathetic account by a wellpublished author and journalist, whose research in Burma included interviews with government leaders.)

project in t.l.e

hi, sir good day,...,,.by the way im danica mae n. grate from second year A,..,.,. im your student in biology and act or t.l.e  thats all and =god bless

               danica mae swufa grate;

hi,,,,,, sir good afternoon by the way im your student in biology and t.l.e
im danica mae grate thats all lang sirrrr thank youisidromax@hotmail.com

hi,,,,, sir  good afternoon by  the way im danica mae grate and my email address in blogspot is danicamaegrate@yahoo.co thank you and god bless sir